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Copper-deficient rat embryos are characterized by low superoxide dismutase activity and elevated superoxide anions

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This interaction triggers a series of defense responses, including the production of ROS and reactive nitrogen species (RNS) and the induction of enzymes that protect against oxidative stress also triggers the synthesis and build-up of signaling molecules like JA, SA, NO, and ET within the cell, thereby influencing the expression of genes related to secondary metabolite production